Male mice defective in the DNA mismatch repair gene PMS2 exhibit abnormal chromosome synapsis in meiosis

نویسندگان

  • Sean M Baker
  • C.Eric Bronner
  • Lin Zhang
  • Annemieke W Plug
  • Merrilee Robatzek
  • Gwynedd Warren
  • Eileen A Elliott
  • Jian Yu
  • Terry Ashley
  • Norman Arnheim
  • Richard A Flavell
  • R.Michael Liskay
چکیده

Using gene targeting in embryonic stem cells, we have derived mice with a null mutation in a DNA mismatch repair gene homolog, PMS2. We observed microsatellite instability in the male germline, in tail, and in tumor DNA of PMS2-deficient animals. We therefore conclude that PMS2 is involved in DNA mismatch repair in a variety of tissues. PMS2-deficient animals appear prone to sarcomas and lymphomas. PMS2-deficient males are infertile, producing only abnormal spermatozoa. Analysis of axial element and synaptonemal complex formation during prophase of meiosis I indicates abnormalities in chromosome synapsis. These observations suggest links among mismatch repair, genetic recombination, and chromosome synapsis in meiosis.

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عنوان ژورنال:
  • Cell

دوره 82  شماره 

صفحات  -

تاریخ انتشار 1995